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Gossamer, it is good that you do not blindly follow the science crowd. But with the way JaffaCake made it real easy by saying the following, I was hoping you would do better on that one!
On the previous page I linked to the human and chimp genomes that are in the NCBI database showing chromosomes with markers at HOX genes which help control limb structure. And hope I can nominate Sanguis (welcome to the forum!) to figure out how to get markers on some of the ERV's or something so that none here have to take the word of someone else they can see it in as much detail as they want.
And I liked the picture of the chromosomes and the links to the NCBI genomes so much I added my response to you into the subsection of the theory that has Chromosomal Adam and Eve in it then rewrote some so it will all together explain more detail about what I think is happening. So at least you helped advance the intelligence theory! Thanks for that. I'll post the improved explanation of the very conclusive evidence that cannot be ignored by changing the subject to what none yet know. We must all honestly focus on what science does very clearly show, then go from there. So here's what I have so far. If you have any scientific evidence to the contrary or adds detail then I will change accordingly.
From:
http://theoryofid.blogspot.com/
Chromosome Speciation (fast - Human, Fruit Fly, Mosquito)
There is chromosome fission speciation which is the result of a chromosome division where they break apart. Chromosomes are also able to relocate parts of themselves on or in other chromosomes (translocations). And the origin of human chromosome #2 is from our chromosome fusion speciation[17] where two joined to become our second largest which in turn led to the 46 chromosome human genome that only humans have.
Phylogenetic evidence indicates a good number of successful chromosome fusions and rearrangements as well as duplications of whole or parts of chromosomes so that complexity increases by starting with what works then readapting for use in another area of the cell nucleus (not leaving it up to random chance). The intelligence mechanism is here taking good guesses. What worked before in a similar situation is copied. Or memory can be moved around which in 3D changes physical location (in nucleus) addressing characteristics.
In human chromosomal fusion speciation there was a "head-to-head telomeric fusion". Telomeres at each end of the 2 chromosomes likely became sticky, possibly by removal of repeating code that forms a protective layer which makes the ends not-sticky. Then when not-sticky ends are in close enough proximity molecular forces of attraction take over then fusion occurs as shown below in Human Chromosome #2.
Side by side comparison of Human and Chimpanzee chromosomes.
http://www.thetech.org/genetics/ask.php?id=69
When the locations of HOX genes that help determine bone size/shape are highlighted we find that some were involved in the fusion allowing the inference that the rearrangement would cause some immediate morphological change to limb structure[26]:
Human:
http://www.ncbi.nlm.nih.gov/projects/mapview/map_search.cgi?taxid=9606&query=HOX**&qchr=&strain=All
Chimp:
http://www.ncbi.nlm.nih.gov/projects/mapview/map_search.cgi?taxid=9598&query=HOX**&qchr=&strain=All
Even though there was not a significant amount of gene scrambling at the fusion site the rearranging of the chromosome territories alone could produce a noticeable enough of a morphological change that the adults would know there was something special and different about them.
The first fused chromosome is in either allele (mother or father) of the haploid (has one of two sets of chromosomes) germ cell (egg or sperm) that divides down to develop into a 47 chromosome heterozygote (alleles are different not homozygous where allele pair each the same) humanoid that has the human chromosome #2 being expressed along with copy of the two chromosomes with no fusion that provides all that the cell had before so it is not a sudden unsurvivable change. The new fused chromosome is controllable through epigenetic systems to immediately reregulate genes to a successful balance. We now have the first human Chromosome #2.
Next, the fused chromosome replicates in the population as follows:
48 and 48 parents produce a 48 offspring only.
48 and 47 parents produce a 48 or 47 offspring.
47 and 47 parents produce a 48 or 47 or 46 human offspring.
47 and 46 parents produce a 47 or 46 human offspring.
46 and 46 parents produce a 46 human offspring only.
The first 46 chromosome humans who were born to the existing 47 chromosome lineage may have right away been fertile, or at first had sterility problems in which case human chromosome #2 had to first learn to survive to replicate without the unfused chromosomes of the other allele there to help maintain proper cell functions. It would then become increasingly difficult for a 46 to reproduce with 48 and possibly 47 in part because along with the new genome design came a new self-image that made the 48's look "apish" and 47's relatively "unattractive" to 46's.
Where "human" is defined as having the unique 46 chromosome genome design that separates us from 47 and 48 ancestors there was a first human couple in our ancestry that was already fully human. There is here a human man and a woman Chromosomal Adam and Eve who together could only produce 46 chromosome descendants whose children would prefer to be with their own kind as would their children's children through time, all the way from them to us.
You can just say because "that is how the Creator works". And no matter what science discovers in the future "that is how the Creator works" which is a good thing because the best from science is yet to come. Some are following a mystery where there is very real intelligence in where we came from that goes right into consciousness and everything beyond. But it requires readjusting to the idea that the genome of Adam and Eve is in all of us right now, which is better than their not being there anyway.
On the previous page I linked to the human and chimp genomes that are in the NCBI database showing chromosomes with markers at HOX genes which help control limb structure. And hope I can nominate Sanguis (welcome to the forum!) to figure out how to get markers on some of the ERV's or something so that none here have to take the word of someone else they can see it in as much detail as they want.
And I liked the picture of the chromosomes and the links to the NCBI genomes so much I added my response to you into the subsection of the theory that has Chromosomal Adam and Eve in it then rewrote some so it will all together explain more detail about what I think is happening. So at least you helped advance the intelligence theory! Thanks for that. I'll post the improved explanation of the very conclusive evidence that cannot be ignored by changing the subject to what none yet know. We must all honestly focus on what science does very clearly show, then go from there. So here's what I have so far. If you have any scientific evidence to the contrary or adds detail then I will change accordingly.
From:
http://theoryofid.blogspot.com/
Chromosome Speciation (fast - Human, Fruit Fly, Mosquito)
There is chromosome fission speciation which is the result of a chromosome division where they break apart. Chromosomes are also able to relocate parts of themselves on or in other chromosomes (translocations). And the origin of human chromosome #2 is from our chromosome fusion speciation[17] where two joined to become our second largest which in turn led to the 46 chromosome human genome that only humans have.
Phylogenetic evidence indicates a good number of successful chromosome fusions and rearrangements as well as duplications of whole or parts of chromosomes so that complexity increases by starting with what works then readapting for use in another area of the cell nucleus (not leaving it up to random chance). The intelligence mechanism is here taking good guesses. What worked before in a similar situation is copied. Or memory can be moved around which in 3D changes physical location (in nucleus) addressing characteristics.
In human chromosomal fusion speciation there was a "head-to-head telomeric fusion". Telomeres at each end of the 2 chromosomes likely became sticky, possibly by removal of repeating code that forms a protective layer which makes the ends not-sticky. Then when not-sticky ends are in close enough proximity molecular forces of attraction take over then fusion occurs as shown below in Human Chromosome #2.
Side by side comparison of Human and Chimpanzee chromosomes.
http://www.thetech.org/genetics/ask.php?id=69
When the locations of HOX genes that help determine bone size/shape are highlighted we find that some were involved in the fusion allowing the inference that the rearrangement would cause some immediate morphological change to limb structure[26]:
Human:
http://www.ncbi.nlm.nih.gov/projects/mapview/map_search.cgi?taxid=9606&query=HOX**&qchr=&strain=All
Chimp:
http://www.ncbi.nlm.nih.gov/projects/mapview/map_search.cgi?taxid=9598&query=HOX**&qchr=&strain=All
Even though there was not a significant amount of gene scrambling at the fusion site the rearranging of the chromosome territories alone could produce a noticeable enough of a morphological change that the adults would know there was something special and different about them.
The first fused chromosome is in either allele (mother or father) of the haploid (has one of two sets of chromosomes) germ cell (egg or sperm) that divides down to develop into a 47 chromosome heterozygote (alleles are different not homozygous where allele pair each the same) humanoid that has the human chromosome #2 being expressed along with copy of the two chromosomes with no fusion that provides all that the cell had before so it is not a sudden unsurvivable change. The new fused chromosome is controllable through epigenetic systems to immediately reregulate genes to a successful balance. We now have the first human Chromosome #2.
Next, the fused chromosome replicates in the population as follows:
48 and 48 parents produce a 48 offspring only.
48 and 47 parents produce a 48 or 47 offspring.
47 and 47 parents produce a 48 or 47 or 46 human offspring.
47 and 46 parents produce a 47 or 46 human offspring.
46 and 46 parents produce a 46 human offspring only.
The first 46 chromosome humans who were born to the existing 47 chromosome lineage may have right away been fertile, or at first had sterility problems in which case human chromosome #2 had to first learn to survive to replicate without the unfused chromosomes of the other allele there to help maintain proper cell functions. It would then become increasingly difficult for a 46 to reproduce with 48 and possibly 47 in part because along with the new genome design came a new self-image that made the 48's look "apish" and 47's relatively "unattractive" to 46's.
Where "human" is defined as having the unique 46 chromosome genome design that separates us from 47 and 48 ancestors there was a first human couple in our ancestry that was already fully human. There is here a human man and a woman Chromosomal Adam and Eve who together could only produce 46 chromosome descendants whose children would prefer to be with their own kind as would their children's children through time, all the way from them to us.
